Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Then again, our capability to rescue SERT perform in KI midbrain synaptoneurosomes from the inhibition of FAK indicates elevated FAK signaling downstream of your Pro32Pro33 mutant, as confirmed by increased pFAK localization in five-HT synapses. https://joanz097dpz9.wikikali.com/user