Preliminary scientific studies show conolidine may inhibit certain ion channels, lessening neuronal excitability and limiting ache signals. This mechanism is especially relevant in neuropathic ache, where abnormal signaling causes persistent pain. Additionally, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to pain notion. Computational docking scientif... https://bobs988sqn0.blogpixi.com/profile